In search of:
students proficient in programming, machine learning, & big data analysis
The Halladay Lab is part of the fastest growing major at its University, the Neuroscience Program at Santa Clara Univeristy and is housed within the Department of Psychology.
Lindsay Halladay traveled to Aspen, CO to present two invited talks at the Winter Conference on Brain Research. Findings of the NCSB lab identified a neural substrate for alcohol-induced anxiety alleviation. Additional research identfiied a corticostriatal pathway mediating ethanol seeking in the face of adverse consequences.
We're helping reactivate the
Bay Area Chapter for the
Society for Neuroscience!
Interested in becoming a
member? Click to let us know!
And see you in San Diego at SFN!
Lindsay Halladay joins the Editorial Board for the Journal of Neuroscience Research as an Associate Editor.
Research Assistant Malia Belnap featured on Santa Clara's website in "Neuroscience Student Researches Anxiety-Alcoholism Connection"
Halladay lab students Gigi Etem and Hannah Henderson present data at SCU's end of the year Neuroscience event
Lindsay Halladay interview
Malia Belnap featured on SCU Psychology site for her recent achievement as 2018 DeNardo Scholar
Research assistant Malia Belnap
selected for DeNardo Science
Scholar Award, a prestigious two-
year grant to lead a project at
Santa Clara University
Exciting data collected by Gigi Etem, Max Bjorni,
Sohrab Sami, and Hannah Henderson presented at GRC:
Alcohol and the Nervous System
highlighted in article
Research conducted in the Neural Circuits, Systems, & Behavior Lab, headed by Dr. Lindsay Halladay, is focused on understanding the physiological processes by which neural activity produces behavior, and more specifically, how dysfunctional circuitry leads to aberrant behavior. NCSB's objective is to better understand how reward and aversion circuits converge and interact. Using a combination of in vivo electrophysiology, behavior, and genetic tools, the Halladay Lab's ultimate goal is to clarify how stress alters neural signaling and influences circuitry in a way that promotes addictive-like behaviors.